Interestingly, not only the increased phosphorylation of AMPK, but also the decreased phosphorylations of P70S6K (a downstream substrate of mTOR Complex 1) and ULK1 (a downstream target of mTOR Complex 1) mediated by the administration of NaHS after AP induction were significantly suppressed by pre‐treatment of CC (Fig. 6B). Here, RPS6KB1 is linked to alkaline phosphatase measurement.