An acutely nebulized ADRB2 agonist caused significant clearance of EVLW, but did not change airway wall thickness or luminal area in the large generations, suggesting the importance of lung fluid in stable HF patients, and the possibility of ADRB2 agonists as a treatment in improving lung air flows and volumes and clearing EVLW. The gene discussed is ADRB2; the disease is hydrops fetalis.