Indeed, the FUS and TDP‐43 results in ALS/FTD complement findings in familial Alzheimer's and Parkinson's diseases where mutation and overexpression of wild‐type amyloid precursor protein and α‐synuclein involving increased gene dosage cause disease 59, 60, 61, 62, 63. The gene discussed is FUS; the disease is amyotrophic lateral sclerosis.