These findings are consistent with mouse models in which hyposulfatemic, Slc26a1-knockout mice exhibit nephrocalcinosis and increased calcium oxalate kidney stone formation while hyposulfatemic, Slc13a1-knockout mice do not exhibit these manifestations (Dawson et al. 2010; Markovich 2011a,b, 2012). Here, SLC26A1 is linked to nephrocalcinosis.