MDL-1 does not appear to regulate the transcriptional response to infection, but there are likely to be multiple effectors downstream of MDL-1 and ZIP-10, as well as downstream of MXL-1 and MXL-2, that are promoting intracellular pathogen development, while effectors downstream of MML-1 likely inhibit intracellular pathogen development. Here, SLC39A10 is linked to infection.