PDGFRB and acute kidney injury: [52, 53] In this setting, our above data are of interest as preconditioning of rats with 1D11 attenuated AA-induced functional and structural AKI onset (PCr increase, PTEC necrosis, peritubular capillaritis, interstitial inflammation, PDGFRβ+ perivascular cells accumulation and mitochondrial and RE stress in tubular epithelial cells).