GJA1 and acute respiratory distress syndrome: With the facts that endotoxin always played an important role in ALI after AOLT, and the destruction of intestine motility and barriers after liver transplantation lead to endotoxin explosion and inflammatory cytokines over-production, resulting in susceptible organs injuries [29, 30], models of LPS-induced BEAS-2B cells were used to test protective effects of Cx43 inhibition.