AngII infusion could induce endothelial miR-712/miR-205 in AAA, both in vitro and in vivo, and the silencing of miR-712/miR-205 by antisense oligonucleotides could decrease inflammation and the activity of endothelail MMPs, thus preventing AAA development in AngII-infused ApoE−/− mice52. Here, AGT is linked to triple-A syndrome.