NR1H4 and metabolic dysfunction-associated steatotic liver disease: In light of the recent data pointing to important metabolic effects of FXR activation in the intestine,51 the existence of gut‐liver signaling pathways, including the FXR‐FGF15/19 pathway (reviewed in Ferrebee and Dawson30), and the recently described intestinal FXR‐ceramide axis,51 changes in expression and/or function of ileal BA transporters may be relevant for NAFLD/NASH.