After 60 h of infection, at the more acute phase, the bacteria had induced a substantial increase in the expression of genes for most proinflammatory cytokines, including IFN-γ, IL-1β, IL-2, IL-6, IL-8, IL-22, and TNF-α, as well as genes for IL-10 and IL-4. The gene discussed is CXCL8; the disease is infection.