EFNB2 and glioblastoma: We found a dramatic impairment of tumour growth in the EFNB2 knock-down group relative to control, in that vector-transduced G26 formed GBM in all animals (as previously reported (Stricker et al., 2012) whilst EFNB2 knock-down cells gave rise to slower growing tumours in only 2 out of 10 animals, with the other 8 mice remaining tumour-free for >1 year.