These mice are highly susceptible to experimental toxoplasmosis and their phenotype is not recapitulated by mice deficient in nucleic acid sensing (TLR3-/TLR7-/TLR9-deficient) but rather by mice deficient in TLR7/TLR9 and TLR11, highlighting the redundancy of pathogen recognition in those animals108 (Figure 4a). This evidence concerns the gene TLR7 and toxoplasmosis.