In order to delineate the molecular mechanism governing the suppressive effect of AR on CD90 expression, we used two different mesenchymal-like (Figure 3B; defined by E-cadherin, N-cadherin, β1-integrin, and vimentin expression) human HCC cell lines (SKhep1 and Tong; CD90-expressing cells) that stably express AR. This evidence concerns the gene VIM and hepatocellular carcinoma.