Consistent with our finding that oxidative stress mediates EndMT, many of the pathways upregulated by TGF-β+H2O2 were related to fibrosis (for example, hepatic fibrosis / hepatic stellate cell activation), inflammation (for example, leukocyte extravasation signalling) or oxidative stress itself (for example, NRF2-mediated oxidative stress response). This evidence concerns the gene TGFB1 and Hepatic fibrosis.