NFKB1 and ulcerative colitis: These findings suggested that GC could regulate diverse lncRNAs expressions; for example, in NF-κB pathway and during proinflammatory cytokines production, GC could upregulate the negative regulatory lncRNAs expression; meanwhile it suppressed the expression of positive regulatory lncRNAs related to NF-κB pathway, which resulted in ameliorate effect on ulcerative colitis.