These findings are consistent with other research that has linked methylation in these early NR3C1 CpG sites to poor stress response in humans and animal models (Weaver et al., 2004; Oberlander et al., 2008), and to neurobehavioral risks such as premature birth (Lester et al., 2015) and maternal anxiety and depression during pregnancy (Oberlander et al., 2014). The gene discussed is NR3C1; the disease is depressive symptom measurement.