ADAMTSL3 and hypertrophic cardiomyopathy: Analysis of the inversely associated interaction relationship pairs showed that mmu-miR-322-5p was decreased with targets that increased Adamts5, Adamtsl3, Arhgap20, Cacna2d1, Cdon, Dclk1, Fgfr1, Has2, Islr, Slit2, and Sobp; of these, Cacna2d1 was involved in the MAPK signaling and hypertrophic cardiomyopathy pathways and Fgfr1 was involved in p53, PI3K-Akt, and Ras signaling.