CCL2 and ductal breast carcinoma in situ: According to ELISA analysis of 2D cultures, Ca-TAT delivery of huCCL2si1 or huCCL2si2 to either MDA-MB-231 or DCIS.com cells resulted in over a 50% knockdown in CCL2 expression, compared to Ca-TAT peptides complexed to control siRNA (Figure 1A-1B).