The risk of deep vein thrombosis (DVT) is one and a half times higher among patients with tuberculosis (TB) compared to those without TB [1] due to a hypercoagulable state which occurs among patients with TB resulting from endothelial dysfunction due to the mycobacteria, increased fibrinogen, fibrin, tissue plasminogen activator, decreased anti-thrombin III [2, 3] and use of rifampicin especially in the first two weeks following TB treatment initiation [4]. This evidence concerns the gene PLAT and tuberculosis.