Therefore, we aimed to compare BRAF V600E, RET/PTC rearrangements (RET/PTC1 and RET/PTC3), and RAS mutations (NRAS, HRAS, and KRAS) in PTCs and FTCs from an iodine‐rich country (Japan) and an iodine‐deficient country (Vietnam) to clarify the relationship between iodine intake and genetic alterations of well‐differentiated thyroid carcinoma. This evidence concerns the gene KRAS and thyroid gland carcinoma.