PML and acute myeloid leukemia: Furthermore, our study does not support the suggestion that mitral valve lengthening represents a primary phenotypic expression of the disease, as first, there was not any kind of difference in AML or PML lengths between the apparently healthy MYBPC3-Q1061X mutation carriers and controls; and second, the difference in PML length between the G+/LVH+ group and the control group was not significant after indexing for BSA, or when adjusted for BSA, age, gender, LV mass and ejection fraction.