CCNA2 and cervical carcinoma: In the present, we showed that Ary can inhibit cervical cancer cell proliferation through G1/S arrest and initiate cervical cancer cells programmed to death by mitochondrial apoptosis way, therefore activate extracellular signal-regulated kinase (ERK) phosphorylation to elicit cell G1/S phase arrest through cyclin A2 and cyclin dependent kinase (Cdk) 2 upregulation.