Fingrut and Flegrut [73] found that MeJA treatment resulted in inactivation of apoptosis hallmarks (i.e., cells apoptosis mediating by caspase-3 and DNA condensation and fragmentation) and increased the death receptor protein tumor necrosis factor receptor 1 (TNFR1), which is related to extrinsic apoptotic signaling in cancer cells. Here, TNFRSF1A is linked to cancer.