HSP90AB1 and renal fibrosis: We finally selected heat shock protein β (Hsp90β) owning to two factors: it was previously proven to regulate the upstream mediators in multiple cellular signalling cascades through stabilizing and maintaining their activities, and it is the only protein possibly involving renal fibrosis, inflammation and tubular epithelial cell death here; it has been rarely seen in previous investigations of salt-loading-induced renal dysfunction.