Consistently, overexpression of hnRNPA1 in 4T1 cells (SK-4T1(hn)-TCL) also reversed the tumor immunogenicity of TCL-pulsed DCs (SK-4T1(hn)-TCL-pulsed DCs) and the treatment decreased the survival rate of test mice, as compared with mice treated with SK-TCL-pulsed DCs (Figure 6d). The gene discussed is HNRNPA1; the disease is neoplasm.