In this study, treatment with AnK, Feno, or Metf significantly increased membrane expression levels GLUT4 by 1.52–2.20-, 1.98-, or 1.86- fold as compared with the HF group, respectively, implying that the increased membrane GLUT4 contents are enhanced to cause glucose uptake, resulting in a decrease in blood glucose levels. The gene discussed is SLC2A4; the disease is hydrops fetalis.