EZH2 and prostate cancer: This data, supported by a broad analysis of human transcriptome data sets (131 prostate cancers (plus 19 metastases), 146 BCs) indicates that EZH2 expression “follows” the rate of cellular division, is under control of proliferation cues, and “passively” correlates with proliferation and proliferation markers (primarily Ki-67), in order to maintain the cellular level of H3K27me3.