Progeria has catalyzed the field of lamin biology and has created a new avenue for aging research, particularly because progerin, the disease‐causing aberrant lamin A protein, is not only generated in affected children, but is also generated at comparatively lower levels in the vasculature and other tissues of non‐progeria individuals (Gordon et al, 2014b). The gene discussed is LMNA; the disease is progeroid syndrome.