However, absence of C/EBPα results in loss of myeloid identity in transgenic mice with BCR-ABL1-induced CML-like disease and, interestingly, causes erythroleukemia instead, suggesting that CEBPα is essential for BCR-ABL1-positive CML [140]. Here, CEBPA is linked to chronic myelogenous leukemia, BCR-ABL1 positive.