We confirmed the hepatic steatosis and damage by hematoxylin-eosin (H & E) staining and an increase of plasma alanine aminotransferase (ALT) level in either the AC or AH group, while normal lobular architecture and cell structure were observed in the NC group (p = 0.0041 for histological score; and p < 0.0001 for ALT). This evidence concerns the gene GPT and fatty liver disease.