ERBB2 and carcinoma: The proportion of precancerous or early lesions (AAH/AIS/Lepidic) (38.1%) in MET exon 14 skipping tumors was significantly higher than that of EGFR (15.9%), KRAS (5.6%), HER2 (21.9%), BRAF (15%), ALK (2.9%), RET (5.6%), and pan-negative (10.3%) lung adenocarcinomas, indicating that mutant MET alone might be insufficient to transform normal cells but depend on other genetic alterations progressing into more aggressive carcinoma subtypes.