Moreover, NDR2-mediated phosphorylation of Rabin8 supports primary cilia formation [40] (Table 1 and Table 2), which supports a possible role of defective NDR2 signalling in ciliopathy [41,42], since primary cilia, as cell cycle regulated antenna-like sensory structures with a centrosome base, play important roles in signal transduction [43,44] and disease-associated processes [45,46]. This evidence concerns the gene STK38L and ciliopathy.