At 48 hai, the activities of oxidoreductases, cellulose synthases and glycosyltransferases were overrepresented in the R NIL, suggesting that the R NIL responded to the infection with cell wall reinforcement (such as lignification, cellulose synthesis, and cuticular wax production), expression of PR proteins (chitinases, lipid transfer proteins, defensins and thaumatin-like proteins) and antimicrobial metabolites (alkaloids and phytoalexin), and trichothecene detoxification (UGTs, GSTs and ABC transporters). The gene discussed is TMEM37; the disease is infection.