In our previous studies, BP was examined for antihepatocellular carcinoma activity by inducing Nur77 (also known as NR4A1) expression, which led to caspase-3-dependent apoptosis (Harn et al., 2011) and triggered an antiplatelet effect through PDGF reduction (Liu et al., 2011). This evidence concerns the gene NR4A1 and carcinoma.