In another study, exogenous delivery of miR-124a by stereotactic injection of neuronal cell-derived EVs prevented pathological loss of GLT1 protein, an important glutamate transporter, selectively lost in amyotrophic lateral sclerosis, in SOD1 G93A mice the experimental model of ALS (Morel et al., 2013). This evidence concerns the gene SLC1A2 and amyotrophic lateral sclerosis.