RPS6KB1 and ovarian neoplasm: Furthermore, p70S6K overexpression in breast, colon, liver, and ovarian tumors are found to be related to aggressive malignant phenotypes [11, 42–44], suggesting that the role of p70S6K in MCS that we propose in ovarian cancer may be extended to other tumor cell types and thereby p70S6K could be an attractive target for therapeutics.