Radiation not only induces an immunogenic death of the cancer cells, but enhances pro-inflammatory chemokines and vascular adhesion molecules, as well as major histocompatibility class I (MHC-I), natural-killer group 2, member D (NKG2D) ligands and Fas/CD95 expression on the cancer cells that survive, effectively converting the irradiated tumor into an immunogenic hub, where cycles of antigen release and cross-presentation can occur over time [8]. This evidence concerns the gene FAS and neoplasm.