Consequently, while there was a significant increase in the number of Rab4/Rab11 multivesicular endosomes in infected control cells, there was not a significant difference between the number of Rab4/Rab11 multivesicular endosomes before and after infection in pkaapRNAi fat body cells (P=0.0025; Fig. 5G). This evidence concerns the gene RAB11A and infection.