2012). This is because a decrease in CV allows the creation of smaller wavelengths, thus facilitating the initiation and maintenance of reentry subsequent to creation of functional conduction block. Our data suggest that VNS is able to moderately increase CV. This increase can be attributed to the increase or further preservation of the gap junction protein connexin‐43 (Cx‐43) expression in the myocardium (Sabbah 2011). It has been shown in previous studies that VNS is able to preserve the expression of Cx‐43 in a rat model of MI (Wu and Lu 2011). Here, GJA1 is linked to myocardial infarction.