Until now, due to the fact that SLAM and nectin-4 receptors present high homology in their amino acid sequences between monkeys and humans [49], different studies have shown that CDV could potentially become capable of using human cell receptors [52] indicating that CDV infection could be a potential future threat for humans [53], since it appears that so far, zoonotic CDV potential has been controlled by measles vaccination in humans [12]. The gene discussed is SLAMF1; the disease is measles.