Indeed, other primary immunodeficiencies that are selective for the absence of IL17F (autosomal dominant IL-17F deficiency) or lack a response to IL17 (autosomal recessive IL-17RA deficiency) further illustrate the importance of IL-17 production for control of bacterial (i.e. S. aureus) and fungal (i.e. C. albicans) infection [8]. This evidence concerns the gene IL17F and hyperinsulinemic hypoglycemia, familial, 4.