Because in ABC-like DLBCLs the BCR.1 signal is attenuated compared to GCB-like DLBCLs (Figure 6B), it would be interesting to determine if the coactivity of classical Jak/STAT and NF-κB signaling characteristic of ABC-like DLBCL can enhance the BCR.1 gene expression independent of c-Myc [19, 50]. This evidence concerns the gene MYC and aneurysmal bone cyst.