In addition, we investigated the expression of FoxP2 in the different components of the extended amygdala, since alterations in the gene encoding this transcription factor have been associated with language learning deficits in humans (Lai et al. 2001; Haesler et al 2007; reviewed by Fisher and Scharff 2009, Fischer and Hammerschmidt 2011, and French and Fisher 2014), and may contribute (not alone, but combination with other genes) to the development of autism (Park et al. 2014), which implies not only deficits in communication but also in social skills (Bacon and Rappold 2012). The gene discussed is FOXP2; the disease is autism.