We investigated the expression of FoxP2 in the different components of the extended amygdala, since alterations in the gene encoding this transcription factor have been associated with speech and language deficits in humans (reviewed by Marcus and Fisher 2003; Fisher and Scharff 2009, and French and Fisher 2014), and may contribute (not alone, but in combination with other genes) to the development of autism (Park et al. 2014), which implies not only deficits in communication but also in social skills (Bacon and Rappold 2012). Here, FOXP2 is linked to autism.