The resveratrol analog Cmpd1 further support the role of STAT3 in anticancer molecular mechanism of this phytochemical: glioblastoma cells, in particular, harbor aberrantly active STAT3 and the treatment with Cmpd1 suppressed the STAT3 tyrosine705 phosphorylation in a dose-dependent manner, concomitantly with the induction of the STAT3 pserine727 and Erk1/2 (27). The gene discussed is STAT3; the disease is glioblastoma.