NINL and infection: Inactivation of numerous genes that affect molting, such as pan-1 [26], the integrity of the cuticle, including dpy-9, osm-11 [2], and acs-3 [27], or fatty acid metabolism (e.g., fasn-1), is known to provoke the “peptide expression no infection”, or Peni phenotype: an elevation of nlp-29p::gfp expression in the absence of infection [8].