Some examples of the SWI/SNF-PRC2/EZH2 oncogenesis hypothesis include malignant rhabdoid tumors (MRT) in which the SWI/SNF subunit INI1 (a.k.a. hSNF5, SMARCB1, BAF47) is inactivated, and in lymphomas with EZH2 activating mutations. This evidence concerns the gene EZH2 and rhabdoid tumor.