Accordingly, CCL11 could act as: (1) an inflammatory chemokine contributing to the host response to neoplasia, (2) a proangiogenic factor promoting new vessel formation, (3) a general pro-inflammatory factor in response to tissue stress and/or necrosis, and/or (4) an autocrine growth factor released by cancer cells to promote proliferation, migration, and invasion [5, 8, 30]. This evidence concerns the gene CCL11 and cancer.