As in women with polycystic ovarian syndrome (PCOS), relative contributions to hyperandrogenisms of adrenals and ovaries are at times difficult to separate [8], so are in women with LFOR contributions to low T. During steroidogenesis, DHEA is via sulfotransferase converted to DHEAS (its 3β-sulfate), a conversion almost exclusively coded by the SULT2A1 gene. This evidence concerns the gene SULT2A1 and polycystic ovary syndrome.