INS and Insulin resistance: Later studies have also confirmed the above findings, where gestational low protein (6% vs. 20% in control) in rats led to insulin resistance in adult offspring by involving an inadequate insulin‐induced phosphorylation of the Insulin receptor, insulin receptor substrate‐1 (IRS‐1) and Akt substrate of 160 kDa (AS160) as well as an impaired GLUT4 translocation and the corresponding increases in glucose and insulin levels when subjected to an IGTT (Blesson et al., 2014).