Deficiency of the TH1-associated CC chemokine receptor CCR5 [18], resulting from a deletion variant of the CCR5 gene (CCR5Δ32), protected against inflammation-associated mortality in dialysis patients [19] and decreased the risk of developing acute graft-versus-host disease after allogenic hematopoietic stem cell transplantation [20]. The gene discussed is CCR5; the disease is acute graft versus host disease.